My son was suspected to have this so called EIA as well. A prescribed dose of cortisol just made it a lot worse. A terrible period of hell followed for 3 months where the best lung specialists and doctors could not find help him get better with all tests you can think of, including Xrays and CT scans. He tried about all the pumps and names mentioned in this thread. At one stage we had about 5 or 6 pumps, plus nebulizer,, supplements, etc , etc . In the end the specialist gave up and said he had to have pshycological problems causing anxiety and that causes his lungs to close. They wanted to put him on axiety meds. We refused and I started my own research and manged to find out what was wrong with him. Anyway, long story short, below is an interesting article that many of you might find interesting. It certainly helped my son. http://forums.realthyroidhelp.com/viewtopic.php?f=2&t=6184 At minimum, if your doctor isn’t aware that too little thyroid hormone regulation can cause breathing problems, share this special report with him or her. The doctor may use the information not only to relieve your breathing problem, but to help other patients, too. Asthma. Some inhalants patients use to relieve their asthmatic symptoms work by stimulating beta2-adrenergic receptors in the bronchial tubes.[28] Stimulating the receptors dilates the tubes and constricts blood vessels in their lining, which dries mucous secretions. These effects of the inhalants relieve shortness of breath, coughing, and wheezing respiration.[25,p.207] An alternative to using the inhalers is to increase the number of beta2-adrenergic receptors in the bronchial tubes by taking an effective daily dose of thyroid hormone.[33] T3 is especially useful for this purpose. In 1991, Egyptian researchers treated 23 asthmatic children with T3.[29] The children weren't hypothyroid. During the 30 days of treatment, they continued to use their usual anti-asthma drugs as needed, but they reduced their doses as low as they could. The researchers wrote, "They [the children] all reported at the end of the 30 days an obvious subjective improvement of their asthmatic conditions with a decrease in the number of exacerbations. Seven patients stopped their usual anti-asthmatic medicines, being maintained on T3 only and 3 have decreased the amount of bronchodilators needed. A significant improvement of pulmonary function tests was noted in all patients." According to the researchers, "All patients tolerated well the T3 regimen without any adverse effect." They concluded that T3 induced beneficial effects: T3 "proves to be a useful adjuvant to classic anti-asthma therapy, and may reduce the amount of bronchodilators needed." Other researchers reported that patients with asthma became hypothyroid from using iodine-containing expectorants. After becoming hypothyroid, their asthma worsened. The worsening of the asthma was relieved, however, when the patients stopped using the expectorants and their thyroid function became normal again.[27] Another group of researchers took hypothyroid children off thyroid hormone for a month. By the end of the month, the children's bronchial tubes became more reactive to antigens. (Heightened reactivity of the bronchial tubes to antigens, of course, is the basis of allergy-induced asthma.) The children then resumed their thyroid hormone therapy. Their bronchial tubes, however, remained more reactive to antigens for two more months. Finally, after twenty more days, their bronchial tubes became normally reactive to antigens.[26]